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Following imaging, tumours harvested from KP and KPN mice confirmed elevated Nrf2 in tumours expressing heterozygous Nrf2 D29H/+ (Fig

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Notably, the accumulation of genetic mutations (e.g., point mutations, inversions, translocations, deletions, and polyploidy) drives the malignant transformation of cancer cells and facilitates the acquisition of cancer-specific hallmarks, including cell immortality, apoptosis inhibition, drug resistance, angiogenesis, invasiveness, and the activation of metastatic processes [3,4,5,6]

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