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Disclosures: Liam Swain: Nothing to Disclose, Abdel Shaheen: Nothing to Disclose, Meng Wang: Nothing to Disclose, Jennifer Flemming: Nothing to Disclose, Mayur Brahmania: Nothing to Disclose, Juan Abraldes: Gilead: Grant/Research Support, 89bio: Consultant, Boston Pharmaceuticals: Consultant, Novo Nordisk: Consultant, Boehringer Ingelheim: Consultant, Astra Zeneca: Consultant, Agomab: Consultant, Terumo: Speaking and Teaching, Cook: Grant/Research Support, Karen Tang: Nothing to Disclose, Jenny Godley: Nothing to Disclose 1550 ENGINEERED HUMAN LIVER CO-CULTURES TO MODEL HEPATIC DYSFUNCTIONS IN METALD Owen Lally 1 , 1 University of Illinois Chicago Background: Metabolic dysfunction-associated liver disease (MASLD) with increased alcohol intake is characterized by symptoms of both MASLD and alcohol-associated liver disease (ALD), known as MetALD

Table of Contents Toggle Basic Peptide Information These identity data are listed in PubChem

Fatigue reduces exercise, which leads to more muscle loss and weight gain

C-peptide inhibits decidualization in human endometrial stromal cells via GSK3beta-PP1