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Molecular modeling was performed to reveal the key features of functionalized NCs and to evaluate the robustness of the oxime bond upon exposure to the solvent

To clarify the causal relationships and stagedependent roles of these factors, we propose a conceptual framework for AD progression (1):Early AD (causal events): Disruption of cerebral iron homeostasis leads to regional iron accumulation, which primes neurons for ferroptosis through elevated ROS and impaired antioxidant defenses (2).Transitional AD (facilitative events): Ferroptotic stress promotes A aggregation and tau hyperphosphorylation

10.1016/j.cub.2016.02.019 136 ViswanathanV

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