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The buffering of antioxidant species can be accomplished by reduced glutathione (GSH), that is also required for phase II detoxification in which endogenous and exogenous toxic metabolites are conjugated to GSH for their removal [10], [11]

[74] noted that exposure rates to APAP in the pediatric population now approach 95%, a factor that precludes the identification of APAP as a causative agent in neurodevelopmental disorders using a multivariate analysis of large data sets [2]

2), the additive effect of having \(\left[ a \right] \& \left[ b \right]\) can be modeled via the concept of dose equivalency

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